To verify the dysfunction of ATP synthesis leads rise of Oxidative Phosphorylation to have an apoptotic cell death in the mitochondria. The decline of ATP synthesis is caused by the increase of the Oxidative Phosphorylation.
Specific AIM 3:To determine the ubiquinone analogueswill provide the supplemented with exogenous ubiquinone to complex I to have a normal rate of oxidation. My hypothesis is the supplemented with ubiquinone can reduce apoptosis in the mitochondria.

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